809 replies to this topic

[Galaxyshock]

It's been around 2 years (can't remember exactly tbh) since my last Phenibut dose. I am on 20mg Cipralex just to get through and still have heavy ups and downs. Magnesium, Zinc and B Vitamins all aggravate my symptoms significantly - and I'm roughly 10x more sensitive to stimulants than before my Phenibut dosing. One night of drinking and I'm seriously depressed for a month afterwards (as a result I don't drink anymore obv.). Is Salvia something you would recommend trying in order to up regulate my serotonogenic and more importantly GABA-B system? If so, could I get away with a very low dose quid method?

Thanks!

Oh, looks like you're another one who has suffered for long after quitting Phenibut. How long and what doses did you use? I quit 16 months ago (after 6 months continous use) and still have some symptoms left, but I do have used it few times during recovery. St. John's Wort has helped a lot, I really became alive again and could handle things a lot better. I've read that L-theanine upregulates GABA-B receptors by blocking mGlu receptors, and is overall very helpful compound (unless you respond badly to it). I started recently 600-1200mg a day to aid the hopefully last bits of recovery. St. John's upregulates serotonergic receptors. I doubt you would want to use anything like Salvia if you're very sensitive to just simple minerals. I mean if you have a bad trip and it leads to massive panic and dp/dr issues etc..

Edited by Galaxyshock, 26 August 2013 - 02:01 AM.

[Super Neuro]

I don't suffer from anxiety, but have many friends who do. One of which, "cycles" diazepam (one pill a week, as diazepam has a half-life of 3 days) and says he hasn't gained any tolerance. Is he fooling himself?

Also, this whole deal with cycling got me thinking: For those with severe anxiety, wouldn't TRT or steroids be a viable option?

[nowayout]

I don't suffer from anxiety, but have many friends who do. One of which, "cycles" diazepam (one pill a week, as diazepam has a half-life of 3 days) and says he hasn't gained any tolerance. Is he fooling himself?

Also, this whole deal with cycling got me thinking: For those with severe anxiety, wouldn't TRT or steroids be a viable option?

He is probably right. One pill a week seems unlikely to cause tolerance. But doesn't he get rebound anxiety towards the end of the week? Benzos can be bad that way.

Low testosterone can indeed cause anxiety and depression and the cure might be as simple as having testosterone replacement. However, testosterone therapy itself can cause anxiety if not done right. Too much testosterone (or certain other androgenic steroids) will have anxiety as a side effect in prone individuals. Too much DHT or estrogens (both downstream metabolites of testosterone) have also been linked to anxiety. Indeed, the link between estrogens and anxiety may be related to the fact that women are more prone to anxiety disorders than men.

This reminds of of something. People suffering from anxiety should definitely have a hormone panel and in particular check estradiol levels. Curing the anxiety might be as simple as taking a low dose of an aromatase inhibitor if estradiol is elevated.

Edited by nowayout, 26 August 2013 - 05:21 AM.

[Sasha_]

I'm taking St John's Wort, Theanine and Magnesium for anxiety (especially social phobia) atm. Now I would like to start taking the substances below as well to increase the effectiveness. Is it safe? Is it beneficial?

1) GARUM ARMORICUM (brands: STABILIUM; ADAPTON)
4) RHODIOLA ROSEA
5) RELORA (MAGNOLIA OFFICINALIS and PHELLODENDRON AMURENSE)

Good you mention Rhodiola since I have found it to be very synergistic when taken simultaneously with St John's Wort, only thing is you really need to cycle this stack, like one week on, one week off. I eventually ditch that stack, although it really helped me in stressfull times. Today I find the ciltep stack (forskolin/artichoke) to help me a lot with self confidence, sometimes I even just take forskolin on its own, with B vitamins, cold shower,
work out , and a meat rich diet (kinda slow carb).works for me.

I decided not to purchase Garum Armoricum as it was both expensive and not many reviews could be found. Most importantly though, why you need to cycle this stack??? I thought they were supposed to be used long term without cycling. This should be the case at least with St. John's Wort, Theanine, Magnesium and Rhodiola Rosea. I don't know about Relora.

You are right for all of the above except for Rhodiola, which cannot be taken chronically if you want it to be effective. Not only have I read that almost everywhere
but I did try to take it without interruption and it eventually stops working and some days may even make you feel drained rather than energetic.
As for St John's Wort, I don't see any need to cycle it, but be warned that its long term use can have bad consequences for your eyes:

Phototoxicity in human retinal pigment epithelial cells promoted by hypericin, a component of St. John's wort.

Wielgus AR, Chignell CF, Miller DS, Van Houten B, Meyer J, Hu DN, Roberts JE.

Source

Laboratory of Pharmacology and Chemistry, National Institute of Environmental Health Sciences, Research Triangle Park, NC, USA.

Abstract


St. John's wort (SJW), an over-the-counter antidepressant, contains hypericin, which absorbs light in the UV and visible ranges. In vivo studies have determined that hypericin is phototoxic to skin and our previous in vitro studies with lens tissues have determined that it is potentially phototoxic to the human lens. To determine if hypericin might also be phototoxic to the human retina, we exposed human retinal pigment epithelial (hRPE) cells to 10(-7) to 10(-5) M hypericin. Fluorescence emission detected from the cells (lambda(ex) = 488 nm; lambda(em) = 505 nm) confirmed hypericin uptake by human RPE. Neither hypericin exposure alone nor visible light exposure alone reduced cell viability. However when irradiated with 0.7 J cm(-2) of visible light (lambda > 400 nm) there was loss of cell viability as measured by MTS and lactate dehydrogenase assays. The presence of hypericin in irradiated hRPE cells significantly changed the redox equilibrium of glutathione and a decrease in the activity of glutathione reductase. Increased lipid peroxidation as measured by the thiobarbituric acid reactive substances assay correlated to hypericin concentration in hRPE cells and visible light radiation. Thus, ingested SJW is potentially phototoxic to the retina and could contribute to retinal or early macular degeneration. PMID: 17576381 [PubMed - indexed for MEDLINE] PMCID: PMC2092452

[BlueCloud]

1) GARUM ARMORICUM (brand: STABILIUM) Absolutely ineffective. Tried 1 box of Stabilium as directed. Nothing.

I also found it completely ineffective. Went thru a whole box.

[Elus]

Effects' class='bbc_url' title='External link' rel='nofollow external'>http://www.ncbi.nlm.nih.gov/pubmed/23107346']Effects of L-theanine or caffeine intake on changes in blood pressure under physical and psychological stresses.

L-theanine, an amino acid contained in green tea leaves, is known to block the binding of L-glutamic acid to glutamate receptors in the brain, and has been considered to cause anti-stress effects by inhibiting cortical neuron excitation. Both L-theanine and caffeine, which green tea contains, have been highlighted for their beneficial effects on cognition and mood.

METHODS: In this study, we investigated the effects of orally administered L-theanine or caffeine on mental task performance and physiological activities under conditions of physical or psychological stress in humans. Fourteen participants each underwent three separate trials, in which they orally took either L-theanine + placebo, caffeine + placebo, or placebo only.

RESULTS: The results after the mental tasks showed that L-theanine significantly inhibited the blood-pressure increases in a high-response group, which consisted of participants whose blood pressure increased more than average by a performance of a mental task after placebo intake. Caffeine tended to have a similar but smaller inhibition of the blood-pressure increases caused by the mental tasks. The result of the Profile of Mood States after the mental tasks also showed that L-theanine reduced the Tension-Anxiety scores as compared with placebo intake.

CONCLUSIONS: The findings above denote that L-theanine not only reduces anxiety but also attenuates the blood-pressure increase in high-stress-response adults.

→ source (external link)

Edited by Elus, 30 August 2013 - 11:50 PM.

[UniQuell]

Wow. A truly amazing and in depth post. As far as lemon balm goes though I'd say it's far far preferable to others like phenibut or picamilon. At least we're talking about preventing breakdown in the case of lemon balm, rather than opening the floodgates and dumping GABA in like with phenibut.

[highchief]

- Beer . Yes , beer , and only beer. I very rarely drink any alcohol, and it usually just makes me sleepy and ill if I have more than one glass. But beer ( and only certain brands, lol..) has an unusual anxiolytic effect, if I don't exceed one glass. And I don't think it's because ( or only because ) of the alcohol, wich leads me to the following list of things I have yet to try :

which brands of beer? I've had similar reactions as of late; most alcoholic drinks makes me sleepy, beer usually I can tolerate.

[machete234]

Alcohol is just horrible in pretty much all dosages and it messes with my sleep so that i wake up at 4 in the morning when I went to bed at 12 drunk.

If I drink some cannabis drink like hash coacoa or something I will be fine all evening, go to bed and sleep and the next day I feel nothing or very little.
Non-poisonous drugs for me from now on.
I will throw my phenibut away too because it sucks, its GHB for the poor.

Edited by machete234, 06 September 2013 - 09:39 AM.

[gregor123456]

Has anyone tried this regimen for a certain amount of time, with significant benefits?

[KimberCT]

Just a quick comment, Relora appears to be mentioned several times in this thread. I didn't see mention that its active constituents, honokiol and magnolol, are PAMs at benzodiazepine sensitive and non-benzodiazepine GABA-A receptors.

Personally, I'm looking for ways to modulate extrasynaptic α4βδ receptors. They're the most complicated I've tried to research though.

[chemicalambrosia]

Just a quick comment, Relora appears to be mentioned several times in this thread. I didn't see mention that its active constituents, honokiol and magnolol, are PAMs at benzodiazepine sensitive and non-benzodiazepine GABA-A receptors.

Personally, I'm looking for ways to modulate extrasynaptic α4βδ receptors. They're the most complicated I've tried to research though.

Interesting, do you have any links for that? I'm sure science guy would like to take Relora out of the recommended section if that is the case(and if he can find the time).

[KimberCT]

Just a quick comment, Relora appears to be mentioned several times in this thread. I didn't see mention that its active constituents, honokiol and magnolol, are PAMs at benzodiazepine sensitive and non-benzodiazepine GABA-A receptors.

Personally, I'm looking for ways to modulate extrasynaptic a4bδ receptors. They're the most complicated I've tried to research though.

Interesting, do you have any links for that? I'm sure science guy would like to take Relora out of the recommended section if that is the case(and if he can find the time).

http://www.ncbi.nlm....les/PMC3652012/

[chemicalambrosia]

Just a quick comment, Relora appears to be mentioned several times in this thread. I didn't see mention that its active constituents, honokiol and magnolol, are PAMs at benzodiazepine sensitive and non-benzodiazepine GABA-A receptors.

Personally, I'm looking for ways to modulate extrasynaptic a4bδ receptors. They're the most complicated I've tried to research though.

Interesting, do you have any links for that? I'm sure science guy would like to take Relora out of the recommended section if that is the case(and if he can find the time).

http://www.ncbi.nlm....les/PMC3652012/

A quote from the end:
"The use of CAM continues to be common among US adults and nearly $15 billion was spent for non-vitamin, non-mineral, natural products in 2007 (Barnes et al., 2008). Our results suggest that magnolia bark extracts or the purified active ingredients magnolol and honokiol would be effective sedatives and anxiolytics. However, as potent and effective modulators of GABA_A receptors, these compounds also have the potential for serious safety concerns, including side-effects and drug interactions. Further studies are needed to determine the effects of co-administration of these compounds with alcohol and with prescription medications, such as the benzodiazepines, which are commonly used to treat insomnia and anxiety. The possibility that magnolol and honokiol may become physically addictive, induce cross-tolerance, or produce withdrawal symptoms after chronic use should also be carefully examined."

Relora probably isn't the thing to use for benzo withdrawal...

[vtrader]

Tried one of those ginseng and bee pollen liquid vial types, bad mistake. I was making progress, but still felt tired by lunch time, this ginseng shot my anxiety into over drive, all the nasty symptoms occured.

I felt alert, but not productive as I was to busy becoming disassocited, unfocused, feeling sick and nervous as hell.
I thought ginseng was meant to be something useful to treat anxiety, but instead it just fired it off quickly.

This does mean now that supps that use to work in giving me energy and focus(green tea, Alcar, l-tyrosine etc) I can never ever use them ever again?

Edited by vtrader, 16 October 2013 - 04:25 PM.

[KimberCT]

Bee pollen and royal jelly give me terrible anxiety. It may not have been the ginseng.

[robosapiens]

Also take note of this strategy, and its anecdotal results http://www.longecity...anita-muscaria/

[woleile]

I am evaluating Methylcobalamin right now.... This is what I have experienced.

5000mcg= (anxiolytic)
10000mcg=(anxiogenic)

For some individuals (i.e. OVERMETHYLATED) even doses just above the RDA will trigger ANXIOGENIC effects, however for others (i.e. UNDERMETHYLATED) it will be much higher doses, like yourself.

Also, for the record I am not an advocate of taking MEGA-DOSES / OVER-DOSES of VITAMINS, since there is mounting evidence regards the LONG-TERM effects being in fact very UNHELPFUL

This may be a valid exception to that rule, however. If Brainfogged is undermethylated because of a genetic inability to properly recycle methyl B12, then a higher dose is appropriate, indefinitely. I would also make sure to take methylfolate (in physiologic doses) with it.

[woleile]

Furthermore, there is ever increasing substantiation that EXCESS VITAMIN B12 is CARCINOGENIC (i.e. CAUSES CANCER). For example, see the following:

Sem Hop. 1970 Jul 10;46(31):2170-4.

[Hazards of administering vitamin B12 to cancer patients].
Cancer incidence and mortality after treatment with folic acid and vitamin B12.
Treatment with folic acid plus vitamin B(12) was associated with increased cancer

Circulating folate, vitamin B12, homocysteine, vitamin B12 transport proteins, and risk of prostate cancer: a case-control study, systematic review, and meta-analysis.

CONCLUSION:
Vitamin B(12) and (in cohort studies) folate were associated with increased prostate cancer risk.

I'm responding to a two-year-old post because I just found this thread and am slowly making my way through it. I learned a lot from your original post that was immediately useful, and I thank you for taking the time to gather all that information and make it available here.

It's hard to know what to conclude from the first three studies, since I couldn't find how much of what form of cobalamin was given, and for how long. To further confuse matters, two of them also included treatment with folic acid.

The last study was more informative. The first thing to note is that it measured folate and B12 levels in a blood sample, not amount consumed via diet or supplementation. In fact fewer than 10% of all the men studied had taken any sort of supplement in the month preceding the study, and the proportion was virtually identical in men with and without cancer.

High circulating levels of a nutrient certainly can come from over-supplementing, but they can also come from an inability to use the nutrient in metabolism, e.g. high levels of phenylalanine in PKU. This second possibility has some support in the study, which states:

(H)igh circulating concentrations of holo-haptocorrin were associated with increased risk, and high circulating concentrations of total-transcobalamin with decreased risk of prostate cancer. [snip] [T]he plausibility of reverse-causation as an explanation for our findings is perhaps reinforced by the absence of an association of holo-transcobalamin, representing the bioavailable fraction of B12, with prostate cancer risk.

What the men with prostate cancer had more of is B12 bound to haptocorrin, a transport/storage molecule that cannot be incorporated into cells. What they did not have more of was bio-available B12; in fact levels of that were virtually identical between cases and controls. The authors conclude in the final paragraph that their findings could indicate a reverse connection (the cancer caused the high B12), and they give several possible mechanisms for this.The most they have to say regarding the possibility of high B12 causing prostate cancer is that their study didn't completely eliminate it.


J Clin Pathol. 1993 Jun;46(6):537-9.
Correlations between holo-transcobalamin II, holo-haptocorrin, and total B12 in serum samples from healthy subjects and patients.

Department of Haematology, St Mary's Hospital Medical School, Imperial College of Science, Technology and Medicine, Norfolk, Lon

CONCLUSIONS:

The total serum B12 concentration is a relatively poor indicator of holo-TCII concentrations and, therefore, of the ability of serum to deliver B12 to tissues.

[Sciencyst]

Regarding lowering cortisol: I read today that armed forces members with lower cortisol levels were more prone to PTSD, and it was mentioned that lower cortisol can make one respond worse to trauma. This may have more to do with an intolerance or sensitivity to stress hormones, however.
Two highly interesting potential anxiolytics I have come across in the past few days: rosemarinic acid, and apigenin.

Rosemarinic acid is an active in rosemary and basil. It acts by inhibiting a specific subtype of GABA transaminase, thereby increasing GABA. I know of at least one pharmaceutical with the same mechanism. http://en.wikipedia....Rosmarinic_acid

Apigenin, the principal constituent of chamomile and also found in Bacopa and certain vegetables, is a benzodiazepine receptor agonist with a half life of over 90 hours!!!! But weak enough to not induce blackouts and phenazepam benders. It is also claimed to be a monoamine transporter activator. I am unfamiliar with such a thing, but it sounds exciting http://en.wikipedia.org/wiki/Apigenin

Oh another one worth mentioning is oleamide. It is a GABA modulator, which sounds quite attractive. It also has affinity towards CB1 receptors and is produced in the body, and in excess when one is sleep deprived. It was put in with synthetic cannabis to counter the extreme anxiogenesis of synthetic cannabinoids, according to observations on other message boards. It seems to be better for sleep than anxiety though.

Edited by katuskoti, 25 November 2013 - 10:31 AM.

[addx]

cortisol is a "stress hormone" - meaning it handles stress, it does not cause it.

the public and media are constantly confusing matters around cortisol and it is incredible how many ignorants conclude that one should simply lower stress hormons by force.

cortisol should not be lowered in any way except to reduce stress that causes it. reducing cortisol will make the stress worse - it will immediately deplete "energy reserves"(infact it will reduce the speed at which energy is produced) and make you feel unable to cope with stress - overwhelmed.

anyone who ever took replacement cortisol(cortef) will tell you exactly the same. cortisol relaxes you infact.

the fact cortisol almost dissappears at night is infact the reason why one feels insomnia at nights, why paranoia increases during night time. i can literally feel that last cortisol spike around midnight and right after it i grow hungry. last cortisol spike is my last chance to sleep... after that i need pharmacology.

the media is completely retarded regarding cortisol information. that's why it's possible for people to actually think that low cortisol will help against PTSD, there's nothing more wrong than that. cortisol is anti-stress.

the problem with cortisol is that long term exposure is not healthy(cushings). it causes high blood sugar and vasoconstriction of periphery - increased abdominal and upper body fat(buffalo hump) etc.

so, stress should be removed. removing cortisol will infact increase stress.

removing cortisol might not cause high blood sugar via cortisol, but it will cause insane sugar spikes via adrenaline which will be used to make up the lack of cortisol - it will not work nearly as good.

[hani]

addx, your post needs some references.

[hullcrush]

I'm on my phone. Comments: Mg is a suspected bzd site ag reversible by flumenazil.
Keep in mind L-Theanine increases your risk for withdrawal seizures even though literature states mixed effects. Grand mal seizure prevention > twitching fasciculation ameloriation.

Thirdly, specific to my case. Aromatase inhibitors lowered my E2 but left GABA upregulated. Progesterone sites decreased, and my seizure threshold is nothing now, reversible by dermal prog.

[AVerwest]

(((excuse me for my bad english guys. it's not my first language.)))

I'm so grateful for this thread and all the research + valuable information it contains.

In my case, I'm about to start a benzo withdrawal program after taking 1mg daily of Xanax XR version for almost 3 years... I don't feel addicted to it, but I must admit it is quite scary to read about so many people struggling when they stop taking it... since I'm taking the extended release version, I don't really have the option to taper off super slowly since I can't break the pills and 0.5mg is the smallest dose of it available.

A psychiatrist suggested me that I could take Magnesium Glycinate (no specified dose here) + Multi vit mainly rich in B complex but with no 'super %s' + Gingko Biloba up to 240mg daily to ease my anxiety levels and panic attacks both DURING the withdrawal while still obviously taking the Xanax XR and also AFTER it.

I would love to get some opinions on this, since there seems to be so much many more options to take while the process lasts and after it too. I've read about Fish Oil, Bacopa and many more of the options which are also listed in this thread... wonder if including one of them, or many, could help me further.

I'm aware I should be attacking my symptoms and state with actual physical effort and I'm doing so (CBT, exercising, etc.) but having a tolerable withdrawal process would be pretty awesome and if some products could help me with that, the better of course!

Edited by AVerwest, 28 November 2013 - 03:01 AM.

[KimberCT]

I'd highly recommend switching to 20mg of diazepam and then tapering off that. My doctor was kind enough to prescribe 10, 5, and 2mg tablets for my taper. Tapering directly off a short acting benzodiazepine like Xanax is just unnecessarily difficult.

[AVerwest]

I'd highly recommend switching to 20mg of diazepam and then tapering off that. My doctor was kind enough to prescribe 10, 5, and 2mg tablets for my taper. Tapering directly off a short acting benzodiazepine like Xanax is just unnecessarily difficult.

Thanks for your answer.

I've definitely read about the option of doing such switches between different benzos in order to achieve a better taper off but I must confess it sort of makes me a bit insecure!

For example, before settling with Xanax XR (alprazolam) I was originally prescribed Clonazepam and I just reacted terribly to it... as a matter of fact and, as crazy as it sounds, I think I had my first strong full blown panic attack after taking Clonazepam for the first time (before any benzo whatsoever). So, there's that... I'm not sure if I want to risk switching to diazepam and maybe feeling extremely weird or worse and then have a tougher time. Maybe what I'm saying makes little sense though :P I think I will speak to the psychiatrist about this option.

It is important to say, though, that I have been taking (and continue to do so) the XR version of Xanax in 1mg, like I said before. The psychiatrist told me that I had less chances of developing tough withdrawal symptoms based on this (the fact that I'm taking the XR version) and that people on the regular alprazolam/Xanax are generally the ones who struggle more with them. maybe this could be a reason why he sounded confident about just tapering off it without mentioning switches to other benzos.

[nowayout]

I'd highly recommend switching to 20mg of diazepam and then tapering off that. My doctor was kind enough to prescribe 10, 5, and 2mg tablets for my taper. Tapering directly off a short acting benzodiazepine like Xanax is just unnecessarily difficult.

Thanks for your answer.

I've definitely read about the option of doing such switches between different benzos in order to achieve a better taper off but I must confess it sort of makes me a bit insecure!

For example, before settling with Xanax XR (alprazolam) I was originally prescribed Clonazepam and I just reacted terribly to it... as a matter of fact and, as crazy as it sounds, I think I had my first strong full blown panic attack after taking Clonazepam for the first time (before any benzo whatsoever). So, there's that... I'm not sure if I want to risk switching to diazepam and maybe feeling extremely weird or worse and then have a tougher time. Maybe what I'm saying makes little sense though :P I think I will speak to the psychiatrist about this option.

It is important to say, though, that I have been taking (and continue to do so) the XR version of Xanax in 1mg, like I said before. The psychiatrist told me that I had less chances of developing tough withdrawal symptoms based on this (the fact that I'm taking the XR version) and that people on the regular alprazolam/Xanax are generally the ones who struggle more with them. maybe this could be a reason why he sounded confident about just tapering off it without mentioning switches to other benzos.

He thought you were on a short acting formulation. The rationale for switching falls away for someone like you who is already on a long-acting formulation. Except - can you get lower doses prescribed, or can you cut them with a pill divider? (I suppose it would depend on what kind of XR technology they employ in your pills.) Otherwise you could also get regular Xanax, that can be divided, for your taper and take them at shorter intervals.

[AVerwest]

Hi there nowayout,

Xanax XR only comes in 2mg, 1mg and 0.5mg versions. I am on 1mg, and have been for almost 3yrs now. This year has been the worst anxiety wise and I attribute it to the fact that I feel very conxious about the fact that I really believe I need to stop this med soon... I'm definitely scared of carrying on on it and later feeling even worse when I have to quit.

The pills can't be broken because it affects the time release of the substance so that really isn't an option apparently.

The psychiatrist was very straightforward with me and he described me a withdrawal plan that lasted 60 days, with dosage changes every 15 days, like this:
-first 15 days: 1mg and 0.5mg on alternate days
-next 15 days: 0.5mg every day
-next 15 days: 0.5mg on alternate days
-next 15 days: 0.5mg once every 3 days
stop

(that's just the plan he suggested me to follow, not intended to say it works or will work for everyone since I haven't even started it yet)

Before I start with it, I'm really wondering which other product I could take during the withdrawal process to ease the anxiety so I'm very curious about all the products suggested in this thread. He suggested Gingko Biloba, omega 3 fatty acids and even Chamomile tea (which is in red list in this thread) so, yeah, some doubts before I start with this...

[KimberCT]

How extended is the XR version? I just really doubt it'd come close to that of diazepam. If you can straight taper, that'd be ideal but diazepam worked better for me.

For what it's worth, my first panic attack occurred on my second day of Xanax. I always noticed there was a short period of time when, as the drug was beginning to kick in, my anxiety would shoot up. This window combined with the rebound anxiety of the prior day's use finally pushed me over the edge.

[KimberCT]

Before I start with it, I'm really wondering which other product I could take during the withdrawal process to ease the anxiety so I'm very curious about all the products suggested in this thread. He suggested Gingko Biloba, omega 3 fatty acids and even Chamomile tea (which is in red list in this thread) so, yeah, some doubts before I start with this...

Oxcarbazepine, nebivolol, l-theanine, and magnesium malate. The latter two require rather frequent, large doses. I prefer nebivolol, although propranolol is more commonly used. I'd avoid ginkgo and high EPA fish oil as both increased my anxiety.