im curious about T-817MA, any more information on this?? as i can see, the post about it is quite old now.
gamesguru, so EGCG and quercetin made you stupid or what, i didnt understand your meaning there
Posted 09 January 2017 - 04:49 AM
im curious about T-817MA, any more information on this?? as i can see, the post about it is quite old now.
gamesguru, so EGCG and quercetin made you stupid or what, i didnt understand your meaning there
Posted 09 January 2017 - 08:14 AM
im curious about T-817MA, any more information on this?? as i can see, the post about it is quite old now.
gamesguru, so EGCG and quercetin made you stupid or what, i didnt understand your meaning there
I think that he was joking, old sport.
PD: Ginkgo is a cheap, highly available source of quercetin.
A standardised extract of Ginkgo biloba leaves is a well defined product and contains approximately 24% flavone glycosides (primarily quercetin, kaempferol and isorhamnetin)
https://www.ncbi.nlm...pubmed/12757407
Edited by William Sterog, 09 January 2017 - 08:16 AM.
Posted 27 December 2017 - 04:23 AM
https://www.hindawi....m/2017/1265464/
Oral Administration of Red Ginseng Extract Promotes Neurorestoration after Compressive Spinal Cord Injury in Rats
Red ginseng and its active ingredients have been shown to decrease neuron death after brain ischemia in experimental animals. However, little is known about the effects of orally administered ginseng extract on spinal cord injury. We orally gave red ginseng extract (RGE) to rats with compressed spinal cord injury (SCI). Open-field locomotor scores were measured as indices of motor function. Histopathological changes and cytokine expressions in situ after SCI were evaluated. Compared to vehicle treatment, RGE treatment (350 mg/kg/day) significantly improved locomotor score up to levels close to those pre-SCI, prevented neuron loss, and facilitated the restoration of white matter in the spinal cord at 14 days after SCI. Treatment with RGE caused less aggregation of Iba-1-positive microglia in grey and white matter at 7 days after SCI, upregulated the expression levels of VEGF and Bcl-xL, and reduced IL-1β and TNFα expressions in the spinal cord at 7 and 14 days after SCI. We concluded that oral administration of RGE facilitates almost complete functional recovery from motor and behavioral abnormalities in rats with SCI and prevents neuron death in situ, possibly through inhibition of inflammation and upregulation of neuroprotective factors in the injured spinal cord.
Posted 29 May 2019 - 07:03 AM
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