162 replies to this topic

[Mind_Paralysis]

If someone is wondering why there's so little documentation on Fasoracetam, then it should probably be added that the main research have been done under the name NS-105 - so it's under that label that you'll find research-info. =) Until Dr Hakonarson comes out with his report next year. If I recall correctly, it'll be sometime in March, most likely.

 

The recommended dosing of Fasoracetam was as high as a 100 mg apparently - according to one of the studies - but the reports here on Longecity all imply that this is waaaay too high of a dose - most people start feeling something at doses as low as 5 mg!

 

The mean dose for those of us that have impairments appear to be somewhat higher tho' - more like 15 - 25 mg's. Some report increased effects all the way up to 50 mg, but hardly anyone seems to feel a difference between 50 and 100 mg's, which is a bit puzzling, but apparently that's a fact. The glutamatergic network obviously reacts differently than individual centers of the brain, when it comes to stimulation.

[eon]

are you saying racemic meth is simply what is technically "street meth"? Would any form of meth have any benefit for the ADHD sufferer? Why couldn't ephedra itself be used? It is the precursor to meth. It used to be available in tea form as well as OTC workout supplement. I think people had intense focus when on it, but so does Yerba Mate tea, which is cleaner than coffee.

 

Just last year a banned OTC workout supplement called CRAZE had a meth-like substance in it, which is said to have hooked the people up on the product as well as intensified the workouts. Reason why it got banned was people tested positive for meth at their workplaces when they swear to their bosses they never used drugs, so the culprit was found in CRAZE.

 

Not sure if just "meth-like" is actually good enough, maybe it isn't up to par with actual meth. The compound found is called "N,alpha-diethylphenylethylamine — has a structure similar to methamphetamine, a powerful, highly addictive, illegal stimulant drug. They believe the new compound is likely less potent than methamphetamine but greater than ephedrine."

 

http://www.usatoday....mpound/2968041/

 

Actually racemic methamphetamine is a schedule II in the US along with dextromethamphetamine. To my knowledge no one manufactures the racemic form.

 

Edited by eon, 05 November 2014 - 07:52 AM.

[eon]

I forgot to ask, methamphetamine vs. amphetamines? I'm reading info. that meth is a neurotoxin while amphetamines such as dextroamphetamine seem more healthier:

 

http://en.wikipedia....xtroamphetamine

 

Dextroamphetamine is used to treat attention deficit hyperactivity disorder (ADHD) and narcolepsy, and is sometimes prescribed off-label for its past medical indications, such as depression, obesity, and nasal congestion.^[10][11] Long-term amphetamine exposure in some animal species is known to produce abnormal dopamine system development or nerve damage,^[12][13] but, in humans with ADHD, amphetamines appear to improve brain development and nerve growth.^[14][15][16] Magnetic resonance imaging studies suggest that long-term treatment with amphetamine decreases abnormalities in brain structure and function found in subjects with ADHD, and improves function in several parts of the brain, such as the right caudate nucleus.^[14][15][16]

 

^{I don't know why anyone would opt for meth (desoxyn) over dextro (dexedrine, etc), is meth simply just on another level which is why it is tightly regulated and rarely prescribed? Which would you rather take? What could be considered the safest and best amphetamine? Would lisdexamfetamine be considered the "lightest" of all amphetamines, if there is such a thing? Dextro be 2nd? Meth be the highest level? I'm a bit confused, but isn't there only 2 types of amphetamines: levo and dextro? Levo is said to be no good, but dextro is what anyone should be on. Where does meth belong to? Meth has both isomers, right? If so, is that why it is a neutoxin because it has both? Which is probably why it is also potent?}

Edited by eon, 05 November 2014 - 09:26 AM.

[Ampamet]

I forgot to ask, methamphetamine vs. amphetamines? I'm reading info. that meth is a neurotoxin while amphetamines such as dextroamphetamine seem more healthier:

 

http://en.wikipedia....xtroamphetamine

 

Dextroamphetamine is used to treat attention deficit hyperactivity disorder (ADHD) and narcolepsy, and is sometimes prescribed off-label for its past medical indications, such as depression, obesity, and nasal congestion.^[10][11] Long-term amphetamine exposure in some animal species is known to produce abnormal dopamine system development or nerve damage,^[12][13] but, in humans with ADHD, amphetamines appear to improve brain development and nerve growth.^[14][15][16] Magnetic resonance imaging studies suggest that long-term treatment with amphetamine decreases abnormalities in brain structure and function found in subjects with ADHD, and improves function in several parts of the brain, such as the right caudate nucleus.^[14][15][16]

 

^{I don't know why anyone would opt for meth (desoxyn) over dextro (dexedrine, etc), is meth simply just on another level which is why it is tightly regulated and rarely prescribed? Which would you rather take? What could be considered the safest and best amphetamine? Would lisdexamfetamine be considered the "lightest" of all amphetamines, if there is such a thing? Dextro be 2nd? Meth be the highest level? I'm a bit confused, but isn't there only 2 types of amphetamines: levo and dextro? Levo is said to be no good, but dextro is what anyone should be on. Where does meth belong to? Meth has both isomers, right? If so, is that why it is a neutoxin because it has both? Which is probably why it is also potent?}

You seem to be a bit confused about different amphetamine-related drugs, haha. There are two seperate chemicals you are talking about here, with several variations thereof. The first is amphetamine, which exists in dextro- and levo- enantiomers. Dextro-amphetamine (dexamfetamine for short) is considered the more CNS active enantiomer of amphetamine, while levo-amphetamine causes more PNS effects. For some, a mix of levo- and dextro- amphetamine provides the most therapeutic effect. For example, adderall is 75% dextro and 25% levo, while street speed is usually 50% levo 50% dextro. This enantiomer distribution is dependent on the process used to make the amphetamine. Others find an enantiopure version of only dexamfetamine to be the most efficacious. This would be dexedrine or vyvanse (lisdexamfetamine, just dexamfetamine bonded to lysine for extended release).

 

Methamphetamine is a seperate chemical, which is an amphetamine molecule with a methyl group attached to it (hence, meth-amphetamine). It, too, has dextro- and levo- enantiomers. These two enantiomers have similar properties to their amphetamine counterparts; dextromethamphetamine is more CNS active and levomethamphetamine is more PNS active.  The methyl group makes methamphetamine more potent and allows it to more quickly pass the BBB. This results in a stronger, more neurotoxic, and more addictive drug than amphetamine. 

 

Both chemicals have showed neurotoxicity in various studies, though I'm not sure if it has been demonstrated in humans. I'm personally of the opinion that amphetamine/methamphetamine use is not sustainable for a long duration, and is instead suited more for short periods of time. Hopefully selective dopamine receptor agonists will provide similar effects to amphetamines while being more sustainable. 

Edited by Ampamet, 05 November 2014 - 05:19 PM.

[eon]

And what could such drug be if they exist? 

 

 

 

Hopefully selective dopamine receptor agonists will provide similar effects to amphetamines while being more sustainable. 

 

[Ampamet]

Something like http://www.ncbi.nlm....pubmed/19020411

[eon]

That link has been published since 2008, I wonder if that chemical is available now? Even if it is available, how does one dose with it when there's no suggested dosage that is tolerable? Start with the lowest dose possible, 1mcg?

Edited by eon, 06 November 2014 - 08:18 AM.

[eon]

N-methylamphetamine is synonimous with meth, is N-methyl-alpha-methylphenethylamine as well or not? Are some similar sounding names like methylphenethylamine close to being meth? 

 

There is a new workout supplement that replaced DMAA called N-Methyltyramine (NMT), curious what is that all about. It's clearly not meth or dexmeth otherwise it would have been banned. It's function is for immune and anti fatigue.

[another_chris]

I recommend all of you checking out my ADHD-thread here on the forums. I try to gather most pertinent information regarding ADHD there.

 

http://www.longecity...ception-thread/

 

As for treating ADHD tho' - my foremost recommendation is to do a GENE-TEST over on 23andme.com, and then report back with the results. Certain genetic errors give different variations of ADHD, and once you know which variation you truly have, then you can medicate accordingly, since different medication are better tolerated by different variations of ADHD.

 

And if you happen to have one of the more common gene-errors... "DRD4 7R allele" - then you probably have an error with your D4 dopamine-receptors. If that's the case, then the research-chemical A-412,997 WILL correct the problem!

 

With far less side-effects than Methylphenidate and Amphetamine -RUBBISH.

Read more about A-412,997 here:

http://en.wikipedia.org/wiki/A-412,997

 

 

And then go over to the fasoracetam -thread, and read up on that compound. It's a racetam currently in serious research for treating ADHD.

 

http://www.longecity...e-6#entry691137

 

Faso appears to increase focus, decrease depression, and improve motivation. The results varies quite a  bit tho', but in general, the side-effects are much, much milder than stimulants - it won't f*ck you up in the same way they do.

 

Hi Stinkorninjor!

 

Have you ever tried A-412,997, yet or is it just theory, that it will help? I never read much about the relation of ADHD-subtypes and genes so far. For myself I only can say, I am tested for ADD (inattentive subtype) and tried methylphenidate which didn't do anything for me in a positive way. With increasing dosage, I started to feel depressed (the depression went away, after taking 5-htp (or waiting some time) but still with no positive effects).

 

Second medication I tried was dexamphetamine, wich helps me a lot with my motivation and not that much for my concentration when it comes to read books for example. Antother positive point is increasing eloquence and I can think more clearly.

 

I am also very interested with your experiences with Fasoracetam if you made some. I must say, it seems that I am a totally non-responder to Racetams, so far. I tried Piraceteam, Phenylpir., Oxyracetam together with choline, minerals, omega3, vitamins etc. So I am sceptical to give Faso a chance, cause it is very expensive, too.

 

Regards, Chris

 

 

[eon]

No mention of Aniracetam? Try that and see.

[eon]

Interesting recent OPINION article on ADHD from the NY Times, seem to make a point that ADHD had its benefits depending on certain time period. While I could not find what the "natural fix" is cosidering the title of the article is "A Natural Fix for ADHD", were they talking about quiting school as a natural fix? Pretty much quitting anything boring will fix ADHD. Is that what the article is saying here? Read on:

 

http://www.nytimes.c...&pgtype=article

[Mind_Paralysis]

 

Interesting recent OPINION article on ADHD from the NY Times, seem to make a point that ADHD had its benefits depending on certain time period. While I could not find what the "natural fix" is cosidering the title of the article is "A Natural Fix for ADHD", were they talking about quiting school as a natural fix? Pretty much quitting anything boring will fix ADHD. Is that what the article is saying here? Read on:

 

http://www.nytimes.c...&pgtype=article

 

 

Interesting article. A bit obvious in hind-sight.

 

The way I read it, is that the authors opinion is that there should be more to ADHD-therapy than medication and behavioural modification, in order to fit EXACTLY into society's needs.

He's saying the needs of the ADHD-population is equally valid, and that those needs can, and should, be a part of the treatment-plan. AND that there are gains, not just for ADHD-people, but for society at large, to accept this, and implement it as part of the treatment.

 

Basically, us ADHD-people need situational advice and specific career-counseling, which could then lead to healthier lives, which would be beneficial to the entirety of the populace, since it could decrease the costs of mental disease, substance-abuse, criminal activity, and so on.

 

He's also saying that medication ISN'T unimportant, but that it should be used in a more natural, considered and AIMED way: improving the lives of people with ADHD, in a way that is non-destructive and non-disruptive. I.e, don't take the meds to live up to certain standards, take the meds when you need to complete a certain section of your life that you absolutely necessarily need augmentation in order to achieve it effectively.

 

It's all quite logical and simple in that regard. And it's something I've been weighing and measuring myself recently, how to view society, and how to socialize with neuro-typicals, as well as what I should expect from myself, in relation to society and interactions with neuro-typicals.
 

[Mind_Paralysis]

 

I recommend all of you checking out my ADHD-thread here on the forums. I try to gather most pertinent information regarding ADHD there.

 

http://www.longecity...ception-thread/

 

As for treating ADHD tho' - my foremost recommendation is to do a GENE-TEST over on 23andme.com, and then report back with the results. Certain genetic errors give different variations of ADHD, and once you know which variation you truly have, then you can medicate accordingly, since different medication are better tolerated by different variations of ADHD.

 

And if you happen to have one of the more common gene-errors... "DRD4 7R allele" - then you probably have an error with your D4 dopamine-receptors. If that's the case, then the research-chemical A-412,997 WILL correct the problem!

 

With far less side-effects than Methylphenidate and Amphetamine -RUBBISH.

Read more about A-412,997 here:

http://en.wikipedia.org/wiki/A-412,997

 

 

And then go over to the fasoracetam -thread, and read up on that compound. It's a racetam currently in serious research for treating ADHD.

 

http://www.longecity...e-6#entry691137

 

Faso appears to increase focus, decrease depression, and improve motivation. The results varies quite a  bit tho', but in general, the side-effects are much, much milder than stimulants - it won't f*ck you up in the same way they do.

 

Hi Stinkorninjor!

 

Have you ever tried A-412,997, yet or is it just theory, that it will help? I never read much about the relation of ADHD-subtypes and genes so far. For myself I only can say, I am tested for ADD (inattentive subtype) and tried methylphenidate which didn't do anything for me in a positive way. With increasing dosage, I started to feel depressed (the depression went away, after taking 5-htp (or waiting some time) but still with no positive effects).

 

Second medication I tried was dexamphetamine, wich helps me a lot with my motivation and not that much for my concentration when it comes to read books for example. Antother positive point is increasing eloquence and I can think more clearly.

 

I am also very interested with your experiences with Fasoracetam if you made some. I must say, it seems that I am a totally non-responder to Racetams, so far. I tried Piraceteam, Phenylpir., Oxyracetam together with choline, minerals, omega3, vitamins etc. So I am sceptical to give Faso a chance, cause it is very expensive, too.

 

Regards, Chris

 

 

 

 

I've never tried A-412,997, so it's all theoretical. The basic science behind it is very encouraging tho', and seems very logical: more accuracy = less problems.

 

The problem lies indeed in that there doesn't seem to be much testing done on human subjects, only test-animals like rats.

None of the trials mention ANY troubling side-effects however, which would be the logical reason for abandoning research on the compound, and not consider it for human trial.

 

So why there is no more testing on the compound is quite puzzling... why NOT pursue such a promising drug?

I mean, ADHD-drugs is a BIG business! And it's growing for every passing years. And developing new drugs would be a way for companies to get new patents, and therefore more revenue.

[eon]

Ampamet, what is your dosage of aniracetam? A book I read suggested 1000mg daily. The book is old information it was published when aniracetam wasn't even available for purchase. A lot of these sellers suggested 750mg 3x daily. If I were to use aniracetam, I would either dose 500mg 2x daily for a maximum of 1000mg. I hope it is effective. I'm still on TMG, b12, Alpha GPC and folate cycle. I wanted to use Aniracetam with Alpha GPC but I don't want to have too much brain supplements with my stack. Is it not a good idea to add aniracetam to my 4 compound stack? I've never heard of anyone doing it.

[Mind_Paralysis]

Aniracetam isn't being researched as treatment against ADHD tho', mate. Fasoracetam however, is. There's a report coming out in March I believe, which will have the results from a study with the aim of using it for that purpose. It's been trialled successfully already by some people here on the boards.

I suggest you try faso instead of Ani, it seems to be the more logical choice.

[AOLministrator]

1) Is it dangerous if I use ritalin at low-moderate doses, no more then 5 days per week?

 

2) Any advice on how to reduce/prevent toxicity? I'm think daily vitamin C and magnesium (memantine is not an option, messes with my memory)

 

3) Can I take racetams on my off days?

 

4) Is Tianeptine a safe option for methyphenidate comedowns? I have used Ritalin before and the comedowns are something I would like to avoid

 

1. No. Though there is a chance you will form a serious drug addictiction to it (since it is essentically just like cocaine, some say it is better some say it is worse for recreational usage), but you always have that chance with other drugs as well as part of your inclination towards addiction, even if the drug isn't right in front of your nose.

 

2. Ritalin is not toxic in normal doages, although it can mentally exhaust people who actually get a non-atypical stimulant effect from it. If you are one of those people, Ritalin is not for you, regardless of your diagnosis except if your aim is to use it like normal people do for studying/motivation or pushing yourself through test, regardless of ADHD .. which also works with corresponding side-effects. You need to take ridiculous amounts for Ritalin to reach toxicity, like huffing 500mg per day or something and even then it will not burn holes in your head, it is mostly just because of excessive dopamine. Though, the risk of stroke is possibly the main concern here.

 

3. Afaik there are zero adverse interactions.

 

4. I am not sure what you mean by 'comedowns' but Ritalin doesn't really have comedowns if you don't abuse it, crank it up too high or huff it (i.e. take your individually ideal therapeutic anti-ADHD dosage). What will happen is that your ADHD comes back when it stops working. If you feel overexerted than you have been actually overexerting yourself on the drug (likely because the drug was pushing you too much). If you huff it or take it on super-empty stomach then you might overshoot your ideal dosage initially because just too much of it worked too soon. Try to avoid that.

Edited by Aolministrator, 11 November 2014 - 01:00 PM.

[eon]

Where do you find out about the updates regarding the clinical trials? Keep us up to date here. I would try it but I haven't read much of what doses people are using but I think you mentioned it before on this thread.

 

Aniracetam isn't being researched as treatment against ADHD tho', mate. Fasoracetam however, is. There's a report coming out in March I believe, which will have the results from a study with the aim of using it for that purpose. It's been trialled successfully already by some people here on the boards.

I suggest you try faso instead of Ani, it seems to be the more logical choice.

 

Edited by eon, 11 November 2014 - 02:23 PM.

[eon]

sleep deprivation promotes BDNF and treats depression? I thought it has caused me depression. Probably because I did it almost regularly. If you think about it, the most powerful people in the world (world leaders, etc) are known to sleep for only a few hours a day. I think I read about Hilary Clinton only sleeping for about under 5 hours (2-3?). I don't have a source but I read it at one point.

 

Regarding sleep deprivation promoting BDNF, is it because you stay up longer "thinking" and being productive as in the case of researchers, pioneers, etc. Or does it have something to do with the actual deprivation of sleep or a combo of both?

 

I know one poster here mentioned fasting to promote BDNF.

[eon]

I'm still deciding if I want to try desoxy (meth) or dexedrine. With me, since it has both levo and dextro, I am hesitating because I did not like the levo that I have tried from the nasal decongestant. I guess dexedrine is the only option here since it only has dextroamphetamine? What other amphetamine has only dextro and none of the levo? I'd prefer low dosage. I think they start at 5mg. The desoxyn only comes in at 5mg, I'm guessing it's because it's the dose with the less side effects and less addictive properties?

[eon]

I'm just reading about memantine working on the D2 receptor. It's also a med for ADHD (perhaps off label use).

 

 

Good to know:
D1 enhances learning

D2 improves focus (less procrastination and less multitasking)
D3 is associated with motor and reward-related behaviour
D4 is associated with novelty seeking; people with this genetic factor tend to be extroverted, quick tempered, impulsive and easily bored.

 

 

[eon]

I'm still willing to try some lithium orotate as it seems to show potential, even according to reviews of people that take them.

 

http://en.wikipedia....Lithium_orotate

 

I'd just have to finish up my racetam cycle, I tend to be obsessed in trying out new compounds all the time. I don't want to mix these up. But I am eyeing lithium.

[Mind_Paralysis]

You're in America mate. Just talk to your doctor about switching to Focalin, and then combine LOW -dose focalin with Intuniv, and you won't get these kinds of side-effects.

Yeah, it ain't ideal, but it's damn near close to it.

Intuniv ( guanfacine) helps with pretty much every side-effect stims give you, and stims help with the few side-effects that Intuniv gives you. The one side-effect I haven't gotten confirmation on, is the appetite-suppressing effect - not sure if Intuniv helps or not.

I get my information regarding the trials of Fasoracetam directly from the researcher who's doing the testing for ADHD right now, btw! = ) I just e-mailed him, and got a reply.

You can do it as well, just google Hakon Hakonarson and you will find his public e-mail for contacting him - he's a member of several faculties and organisations.

 

 

From: hakonarson@xxxx
To: xxx@hotmail.com
Subject: RE: May I ask a few questions, regarding your research?
Date: Mon, 4 Aug 2014 23:39:49 +0000
 

Thank you xxx, we recently published a paper in Nat Communication on the role of mGluR gene networks in Autism (somewhat comparable to what we have seen in ADHD).  We have identified ADHD patients with mutations in this pathway we believe will benefit from taking fasoracetam.  We are in the planning phase of the first clinical trial we will launch this Fall and have the first human trial results in ADHD in the Feb/March 2015 timeframe which will tell us if the drug has efficacy in ADHD, at least in a preliminary way; hope this information is helpful to you.

 

Best

Hakon 

 

[eon]

does 5mg desoxyn produce obvious side effects associated with being on meth or is it more subtle considering the low dose? Anyone here ever used 5mg Desoxyn?

 

I'm thinking if I buy Desoxyn and scrape the pill to fix my dose I think I'd like that. The pill is in 5 mg dosage and I want to try a lower dose instead, say 1 mg? perhaps micrograms?

 

On a side note:

 

There's actually a supplement called BDNF now? Look at this article:

 

http://www.vox.com/2...chology-science

 

Curious what the IGF-II is all about? I think hat stands for insulin growth factor? Or what is the PKMzeta? Some interesting new products...

 

Edited by eon, 14 November 2014 - 08:45 AM.

[eon]

Regarding the suggestion of lithium orotate, there are other lithium forms out there such as arginate:

 

http://www.amazon.co...ologics lithium

 

This product has lower dose of lithium which could be ideal? It's 50mcg compared to the higher dose lithium orotate of 5mg.

 

Lithium:

 

"A mineral considered important for restoring psychological balance, yet toxic if taken long term, in high amounts and in the wrong form."

 

Now this other lithium product below did not state which form it is but stated from vegetable culture and only has 50mcg as well which I think is low dose which is ideal for something like lithium:

 

http://www.pureformu...s-research.html

 

I'm still deciding which one to get or should I stick with orotate which are usually dosed at 5mg.

[TheBatman]

 

Regarding the suggestion of lithium orotate, there are other lithium forms out there such as arginate:

 

http://www.amazon.co...ologics lithium

 

This product has lower dose of lithium which could be ideal? It's 50mcg compared to the higher dose lithium orotate of 5mg.

 

Lithium:

 

"A mineral considered important for restoring psychological balance, yet toxic if taken long term, in high amounts and in the wrong form."

 

Now this other lithium product below did not state which form it is but stated from vegetable culture and only has 50mcg as well which I think is low dose which is ideal for something like lithium:

 

http://www.pureformu...s-research.html

 

I'm still deciding which one to get or should I stick with orotate which are usually dosed at 5mg.

 

 

I used to take lithium orotate.everyday in the morning. I wouldn't recommend taking it more than once a week in small dosages due to its very long elimination half life. I had a bit too much in my system and I felt zombied out while on it, and for weeks after I stopped taking it.  

 

Edited by TheBatman, 21 November 2014 - 04:44 AM.

[eon]

Not sure how you were dosing it but feeling like a zombie sounds like what people described of people on long term meth or amphetamines. I'm thinking 5 mg of lithium orotate is a bit much compared to the 50 mcg lithium products.

 

What's the half life of lithium anyway? Isn't 1 dose enough to simply "supplement" the brain with it per se? I was hoping a single dose of desoxyn would change the brain for the better. There was a saying about cocaine that once you try it, it changes your brain. My assumption is a single dose of meth could somehow "trigger" the brain to not make one ADHD anymore but I'm sure the doctor's suggestion would be to take it so you can get refills and make the pharmaceuticals richer.

Edited by eon, 21 November 2014 - 07:14 AM.

[Mind_Paralysis]

My assumption is a single dose of meth could somehow "trigger" the brain to not make one ADHD anymore but I'm sure the doctor's suggestion would be to take it so you can get refills and make the pharmaceuticals richer.

 

That is a very incorrect assumption. If simply using a stimulant could cure ADHD, then we'd all be cured by now. All Methamphetamine does, is it's more potent than regular amphetamine, so it's more difficult to dose - yet it also means you need less.

 

ADHD is a molecular, genetic disease, in general. You can't change your genes with this kind of medication.

Now, if you are treated as a child, all the way up to adulthood, you DO in general end up with a brain that's more similar to neuro-typicals than the regular ADHD-brain. But it's only on the surface - you'll still need to continously feed your brain extra chemicals, in order to actually BE normal.

 

You brain gets better at handling itself with chronic stimulant-use, but it won't cure you. You'll always be somewhat altered from the typical population.

[eon]

Is procrastination part of having ADHD? I seem to be very good at it.

[eon]

is meth or amphetamines any good for OCD? I would think it is due to it making one have "focus" considering OCD is of intrusive thoughts and pretty much you lose focus. I can't find any info. regarding meth's role in treating OCD.

[Keizo]

is meth or amphetamines any good for OCD? I would think it is due to it making one have "focus" considering OCD is of intrusive thoughts and pretty much you lose focus. I can't find any info. regarding meth's role in treating OCD.

It seems like it can be.

 

I would assume, inferring from my experience with d-amp, that 1.25 or 2.5mg d-meth would be reasonable to start at. Though of course over time higher doses would mellow down.

My main reason for having used the low doses is my heart can't stand this stuff (D-meth should be more mild in this regard), though they do work. 

Edited by Keizo, 22 November 2014 - 06:50 PM.